OSTEOARTHROSIS ; ANCIENT AYURVEDA HELPS TO GET NORMALISING ; WELL EARLIER IF WE SWITCH ON TO AYURVEDA, FOR JOINT DISORDERS IN CARTILAGINOUS SUPPORT AND INFLAMMATORY CHANGES LEADING TO SYNOVIAL EFFUSION IN COMMON. :

 Many osteoarthrosis, tendon stretches, musculoskeletal  problems are well  treated by ancient ayurveda methods. 

Once  a mining worker visited me. He had given resignation letter to his boss,  because of knee joint arthritis which restricted him to work properly. I asked him to take 3-4 months leave by taking back resignation letter. He took treatment and simple diet  after which he was confident to work and he returned to work. He was capable to work well. 

We should not take preserved foods, sambar,  keeping many days mutton, dosa,  idli wet flours in fridge,  should not be used.This causes liver problems which cannot be identified by lab investigations for many years. This causes narrowing arteries and veins especially where in finer circulation exists. Even in heart and kidney also same circulatory changes occurs. This has to be treated early.  Otherwise, it causes irreversible damage in circulatory mechanism which hinders the proper nutrition to joints, liver, kidney, heart etc.This principle has been told many thousands of years ago. Replacement of joints surgery ; when basic things  of body circulation and other  functions are interrupted is a failure. Because all body functions should be corrected ;only part replacement will not work. So earlier going to ayurveda for proper assessment of disease process, food habits, diets in regular habits, the place of living, daily routine work exercises, seasonal food  habits which acts on body ; which body tends to adjust or adopt (immunity );hence changes in life style according to seasons is essential. So early treatment is best  before unrepairable damage ; has good result.So its necessary to try ayurveda for proper treatment.Tastes of drugs in ayurveda may be not be comfortable for many.But it is life saving and  we should make our mind bold to decide.The penetration of drugs action  & result are proportional to diet, follow up for proper absorption, assimilation.  It is a sensitive treatment on particular individual and it differs from individual to individual.           It has been said : It is wise to consider all non-surgical treatment options before resorting to an artificial hip or knee joint implant," said EULAR President Professor Iain B. McInnes, University of Glasgow, Scotland. Low calcium intake contributes to diminished bone density, early bone loss and an increased risk of fractures. Eating disorders. Severely restricting food intake and being underweight; weakens bone in both men and women. There's no cure for osteoporosis, but proper treatment can help protect and strengthen your bones. These treatments can help slow the breakdown of bone in your body, and some treatments can spur the growth of new bone. In addition to causing permanent pain, osteoporosis causes some patients to lose height. When osteoporosis affects vertebrae, or the bones of the spine Weaken.Osteoarthritis is the most commonly diagnosed type of joint arthritis disease, which can affect hands, knees and hips. Knee arthritis is known to affect joint functionality causing knee pain and even leading to disability as it progresses. There are different stages of knee osteoarthritis (OA).The Center for Disease Control and Prevention has found that the number of people suffering from knee pain disorder is gradually rising.OA pain is easily identifiable through diagnostics and common symptoms. Some people who suffer from immense osteoarthritis knee pain may only show mild changes on x-ray.

Lifestyle considerations like regular exercise may also prove to be helpful. Typically, the area around the knee joints will feel stiff and uncomfortable, particularly when sitting for an extended period, after rising in the morning, or after a workout. Though the cartilage and soft tissues remains at a healthy size, there is proteolytic breakdown of the cartilage matrix from an increased production of enzymes, such as metalloproteinase.
When your physician detects and diagnoses OA at this early stage, it is easier to follow a plan to stop the progression of this joint disease. , where there is obvious erosion to the cartilage surface between bones and fibrillation narrows the gap between the bones. There are proteoglycan and collagen fragments released into the synovial fluid as the disease progresses, wherein the bones develop spurs at the joints as it becomes rougher.With the progression of osteoarthritis of the knee, there is obvious joint inflammation which causes frequent pain when walking, running, squatting, extending or kneeling. Along with joint stiffness after sitting for long or when waking up in the morning, there may be popping or snapping sounds.
Weight loss program, typically lasts six months ; which may help a little. Severe OA is considered  when  the joint space between the bones are considerably reduced, causing the cartilage to wear off, leaving the joint stiff. The breakdown of cartilage leads to a chronic inflammatory response, with decreased synovial fluid that causes friction, greater pain and discomfort when walking or moving the joint.
There is increased production of synovial metalloproteinases, cytokines and TNF that can diffuse back into the cartilage to destroy soft tissue around the knee. The advanced stage of the disease shows development of more spurs causing excruciating pain, which makes even everyday chores, including walking and descending stairs a challenge.
In cases of severe OA of the knee, an  orthopedic surgeon cuts the bone above or below the knee to shorten the length and help realign it for less stress on the knee joint. This surgery helps protect the knee by shifting the weight of the body away from the site of the bone spur growth and damage.

Another surgicaloption is total knee replacement,  or  arthroplasty. During this surgical procedure, the damaged joint is removed and replaced with a plastic or metal prosthesis device. Recovery from surgery may take several weeks and requires patience and discipline, with continuous physical and occupational therapy to regain full mobility.

Clinical Nutrition & Dietetics: There are specific recommendations in regard to nutritional supplements and botanical medicines. Osteoarthrosis (OA) is sometimes referred to as osteoarthritis, degenerative arthritis, or degenerative joint disease. It is characterized by degeneration of the articular cartilage and subchondral bone. It can result in joint deformities, with characteristic Bouchard’s nodes in the proximal phalangeal joints of the fingers and Heberden’s nodes in the distal joints of the fingers. The predictable symptoms include joint pain and stiffness, becoming more chronic over time. Other symptoms commonly included joint swelling, weakness and/or paresthesias in the extremities and decreased range of motion. Typically, joints in the fingers, thumb, knees, spine, and hips can be involved. It can also be the result of trauma to a single joint leading to degeneration of the joint years later. Abnormal cartilage, congenital deformities of joint(s), infection of a joint, crystal deposition and/or other arthritic conditions can lead to OA of the joint. The knee is the most common joint to be affected.
Conventional treatment includes non-steroidal anti-inflammatory drugs. While their use is widespread, there is some evidence that long-term use may inhibit the synthesis of the cartilage matrix and accelerate the deterioration of cartilage. However, there is some evidence that is contradictory. There are some studies that demonstrate that the anti-inflammatory effects of low-dose aspirin slow the progression on cartilage loss in OA.  Additionally, there are significant risks associated with the use of NSAIDs; including gastrointestinal bleeding and cardiovascular events.
Musculoskeletal joint derangement and misalignments have been demonstrated to play a role in the development of OA. Joint alignment, muscle strength nutritional status, previous trauma and mid-stance loads all seem to contribute to the development of OA, Swimming and isometrics have been identified as superior to other types of exercise for the treatment of OA.  Conversely, strengthening the muscles controlling the hip and knee is an effective way to mitigate some of the disabling symptoms associated with OA in the lower extremity.
Diet should focus on the decrease of inflammation, as well as improving insulin sensitivity and achieving ideal body weight. Obesity is a major risk factor for the development of OA in the knees and hips. There is considerable evidence linking metabolic syndrome with inflammatory cytokines implicated in OA. Insulin stimulates chondrocytes and chondroblasts to secrete proteoglycans, which are essential to the proper function of cartilage. Decreased sensitivity to insulin has a detrimental effect on cartilage. Combining weight loss and exercise is known to decrease the risk of developing OA, as well as reducing the symptoms of OA and improving function. General guidelines for diet include a plant-based diet, relying on fresh fruits and vegetables, as well as whole grains, fatty fish and lean meats. Dairy products and gluten-containing grains are known to be allergenic in many people, and therefore should be minimized or eliminated from the diet. Certain individuals may benefit from eliminating nightshades (tomatoes, white potatoes, peppers, and eggplant) from the diet, most likely because of an individual’s sensitivity to these plants. One study looked at Diosgenin, a steroidal saponin found in nightshades. Diosgenin possesses diverse biological activities including anti-inflammatory properties. It has been shown to inhibit the synthesis of inflammatory mediators, such as prostaglandin E2 and interleukins. Therefore, there is a possibility that these plants may actually be beneficial for OA.
Antioxidants:The intake of carotenoids, ascorbate and vitamin E have all been demonstrated to improve the symptoms of OA and even to reverse the progression of the disease. The odds of having hip OA were reduced by nearly two fold when the individual consumed recommended or higher amounts of vitamin C. It has been demonstrated that vitamin C protects chondrocytes from oxidative damage from hydrogen peroxide by regulating multiple regulatory pathways. Astaxanthin, a carotenoid found in a variety of marine mammals and plants, has been shown to reduce Matrix Metalloproteinases (MMP), a substance known to degrade cartilage. The carotenoids in saffron, namely crocins and crocetin, are known to prevent the expression of MMP due to IL.Vitamin D:According to several studies, low plasma levels of vitamin D are associated with an increased risk of OA. However, there are some studies that contradict these findings. Improved outcomes following knee arthroplasty were noted after vitamin D supplementation.  Vitamin D has been shown to have a positive effect on the mass of the quadriceps muscles. It seems that inadequate levels of vitamin D contribute to the progression of OA, but it is not clear whether supplementation is beneficial in improving symptoms in those afflicted with the disease.

Vitamin K:Low vitamin K status is associated with an increased incidence of OA. The anti-inflammatory effects of vitamin K are thought to play an important role in the prevention of OA. The carboxylation of osteocalcin is dependent on vitamin K. Vitamin K deficiency caused serum undercarboxyled osteocalcin levels to increase.        

Many Many osteoarthrosis, tendon stretches, musculoskeletal  problems are well  treated by ancient ayurveda methods.once  a mine worker who gave resignation letter to to his boss because of knee joit arthritis , restricted him to work properly, came. I asked him to take 3-4 months leave by taking back resignation letter. He took treatment and simple diet  made himself confident to work.. and returned to work. He said is now capable to work well. We should not take preserved foods, sambar, keeping many days mutton, dosa idli wet flours should not be used.This causes liver problems which cannot be identified by lab investigations for many years. This causes narrowing arteries and veins especially where in finer circulation exists.Even in heart, kidney also same circulatory changes occurs. This has to be treated early. Or otherwise it causes irreversible damage in circulatory mechanism which hinders the proper nutrition to joints, liver, kidney, heart etc.This principle hsa been told many thousands of years ago.replacement of joints when basic things are are interrupted is failure. So earlier going to ayurveda for proper assessment of disease process, food habbits, diets in regular habbits, the place of living, daily routine work exercises, food seasons which acts on body which body tends to adjustor adopt ;hence changes in life style according to seasons is essential. So early treatment is best and has good result. Importance of treatment is necessary  to try properly.Tastes of drugs in ayurveda may be difficult for many.But its life saving and make our mind bold to decide.The penetration of drugs action  result are are proportional. Diet, follow up for proper absorption assimilation  are sensitive treatment on particular individual.                 It has been said : It is wise to consider all non-surgical treatment options before resorting to an artificial hip or knee joint implant," said EULAR President Professor Iain B. McInnes, University of Glasgow, Scotland. Low calcium intake contributes to diminished bone density, early bone loss and an increased risk of fractures. Eating disorders. Severely restricting food intake and being underweight weakens bone in both men and women. There's no cure for osteoporosis, but proper treatment can help protect and strengthen your bones. These treatments can help slow the breakdown of bone in your body, and some treatments can spur the growth of new bone. In addition to causing permanent pain, osteoporosis causes some patients to lose height. When osteoporosis affects vertebrae, or the bones of the spine Weaken.Osteoarthritis is the most commonly diagnosed type of joint arthritis disease, which can affect hands, knees and hips. Knee arthritis is known to affect joint functionality causing knee pain and even leading to disability as it progresses. There are different stages of knee osteoarthritis (OA).The Center for Disease Control and Prevention has found that the number of people suffering from knee pain disorder is gradually rising.OA pain is easily identifiable through diagnostics and common symptoms. Some people who suffer from immense osteoarthritis knee pain may only show mild changes on x-ray

Lifestyle considerations like regular exercise may also prove to be helpful. Typically, the area around the knee joints will feel stiff and uncomfortable, particularly when sitting for an extended period, after rising in the morning, or after a workout. Though the cartilage and soft tissues remains at a healthy size, there is proteolytic breakdown of the cartilage matrix from an increased production of enzymes, such as metalloproteinase.
When your physician detects and diagnoses OA at this early stage, it is easier to follow a plan to stop the progression of this joint disease. , where there is obvious erosion to the cartilage surface between bones and fibrillation narrows the gap between the bones. There are proteoglycan and collagen fragments released into the synovial fluid as the disease progresses, wherein the bones develop spurs at the joints as it becomes rougher.With the progression of osteoarthritis of the knee, there is obvious joint inflammation which causes frequent pain when walking, running, squatting, extending or kneeling. Along with joint stiffness after sitting for long or when waking up in the morning, there may be popping or snapping sounds.
weight loss program, typically lasts six months ; which may help a little. Severe OA is considered  when  the joint space between the bones are considerably reduced, causing the cartilage to wear off, leaving the joint stiff. The breakdown of cartilage leads to a chronic inflammatory response, with decreased synovial fluid that causes friction, greater pain and discomfort when walking or moving the joint.
There is increased production of synovial metalloproteinases, cytokines and TNF that can diffuse back into the cartilage to destroy soft tissue around the knee. The advanced stage of the disease shows development of more spurs causing excruciating pain, which makes even everyday chores, including walking and descending stairs a challenge.
In cases of severe OA of the knee, an  orthopedic surgeon cuts the bone above or below the knee to shorten the length and help realign it for less stress on the knee joint. This surgery helps protect the knee by shifting the weight of the body away from the site of the bone spur growth and bone damage.

Another surgical option is total knee replacement, or arthroplasty. During this surgical procedure, the damaged joint is removed and replaced with a plastic or metal prosthesis device. Recovery from surgery may take several weeks and requires patience and discipline, with continuous physical and occupational therapy to regain full mobility.

Clinical Nutrition & Dietetics:. There are specific recommendations in regard to nutritional supplements and botanical medicines. Osteoarthrosis (OA) is sometimes referred to as osteoarthritis, degenerative arthritis, or degenerative joint disease. It is characterized by degeneration of the articular cartilage and subchondral bone. It can result in joint deformities, with characteristic Bouchard’s nodes in the proximal phalangeal joints of the fingers and Heberden’s nodes in the distal joints of the fingers. The predictable symptoms include joint pain and stiffness, becoming more chronic over time. Other symptoms commonly included joint swelling, weakness and/or paresthesias in the extremities and decreased range of motion. Typically, joints in the fingers, thumb, knees, spine, and hips can be involved. It can also be the result of trauma to a single joint leading to degeneration of the joint years later. Abnormal cartilage, congenital deformities of joint(s), infection of a joint, crystal deposition and/or other arthritic conditions can lead to OA of the joint. The knee is the most common joint to be affected.
Conventional treatment includes non-steroidal anti-inflammatory drugs. While their use is widespread, there is some evidence that long-term use may inhibit the synthesis of the cartilage matrix and accelerate the deterioration of cartilage. However, there is some evidence that is contradictory. There are some studies that demonstrate that the anti-inflammatory effects of low-dose aspirin slow the progression on cartilage loss in OA.  Additionally, there are significant risks associated with the use of NSAIDs; including gastrointestinal bleeding and cardiovascular events.
Musculoskeletal joint derangement and misalignments have been demonstrated to play a role in the development of OA. Joint alignment, muscle strength nutritional status, previous trauma and mid-stance loads all seem to contribute to the development of OA, Swimming and isometrics have been identified as superior to other types of exercise for the treatment of OA.  Conversely, strengthening the muscles controlling the hip and knee is an effective way to mitigate some of the disabling symptoms associated with OA in the lower extremity.
Diet should focus on the decrease of inflammation, as well as improving insulin sensitivity and achieving ideal body weight. Obesity is a major risk factor for the development of OA in the knees and hips. There is considerable evidence linking metabolic syndrome with inflammatory cytokines implicated in OA. Insulin stimulates chondrocytes and chondroblasts to secrete proteoglycans, which are essential to the proper function of cartilage. Decreased sensitivity to insulin has a detrimental effect on cartilage. Combining weight loss and exercise is known to decrease the risk of developing OA, as well as reducing the symptoms of OA and improving function. General guidelines for diet include a plant-based diet, relying on fresh fruits and vegetables, as well as whole grains, fatty fish and lean meats. Dairy products and gluten-containing grains are known to be allergenic in many people, and therefore should be minimized or eliminated from the diet. Certain individuals may benefit from eliminating nightshades (tomatoes, white potatoes, peppers, and eggplant) from the diet, most likely because of an individual’s sensitivity to these plants. One study looked at Diosgenin, a steroidal saponin found in nightshades. Diosgenin possesses diverse biological activities including anti-inflammatory properties. It has been shown to inhibit the synthesis of inflammatory mediators, such as prostaglandin E2 and interleukins. Therefore, there is a possibility that these plants may actually be beneficial for OA.
Antioxidants:The intake of carotenoids, ascorbate and vitamin E have all been demonstrated to improve the symptoms of OA and even to reverse the progression of the disease. The odds of having hip OA were reduced by nearly two fold when the individual consumed recommended or higher amounts of vitamin C. It has been demonstrated that vitamin C protects chondrocytes from oxidative damage from hydrogen peroxide by regulating multiple regulatory pathways. Astaxanthin, a carotenoid found in a variety of marine mammals and plants, has been shown to reduce Matrix Metalloproteinases (MMP), a substance known to degrade cartilage. The carotenoids in saffron, namely crocins and crocetin, are known to prevent the expression of MMP due to IL.Vitamin D:According to several studies, low plasma levels of vitamin D are associated with an increased risk of OA. However, there are some studies that contradict these findings. Improved outcomes following knee arthroplasty were noted after vitamin D supplementation.  Vitamin D has been shown to have a positive effect on the mass of the quadriceps muscles. It seems that inadequate levels of vitamin D contribute to the progression of OA, but it is not clear whether supplementation is beneficial in improving symptoms in those afflicted with the disease.

Vitamin K:Low vitamin K status is associated with an increased incidence of OA. The anti-inflammatory effects of vitamin K are thought to play an important role in the prevention of OA. The carboxylation of osteocalcin is dependent on vitamin K. Vitamin K deficiency caused serum undercarboxyled osteocalcin levels to increase. So in principle of ancient ayurveda way of treatment is broadly considered;in treating well.  I have found cartilaginous erosion injury have been got relieved. Synovial effusion stopped. Joint strengthened. Ayurveda has explained rasayana for nutrition in different diseases and conditions of nececity ;which has got importance in treatment., tendon stretches, musculoskeletal  problems are well  treated by ancient ayurveda methods.once  a mine worker who gave resignation letter to to his boss because of knee joit arthritis , restricted him to work properly, came. I asked him to take 3-4 months leave by taking back resignation letter. He took treatment and simple diet  made himself confident to work.. and returned to work. He said is now capable to work well. We should not take preserved foods, sambar, keeping many days mutton, dosa idli wet flours should not be used.This causes liver problems which cannot be identified by lab investigations for many years. This causes narrowing arteries and veins especially where in finer circulation exists.Even in heart, kidney also same circulatory changes occurs. This has to be treated early. Or otherwise it causes irreversible damage in circulatory mechanism which hinders the proper nutrition to joints, liver, kidney, heart etc.This principle hsa been told many thousands of years ago.replacement of joints when basic things are are interrupted is failure. So earlier going to ayurveda for proper assessment of disease process, food habbits, diets in regular habbits, the place of living, daily routine work exercises, food seasons which acts on body which body tends to adjustor adopt ;hence changes in life style according to seasons is essential. So early treatment is best and has good result. Importance of treatment is necessary  to try properly.Tastes of drugs in ayurveda may be difficult for many.But its life saving and make our mind bold to decide.The penetration of drugs action  result are are proportional. Diet, follow up for proper absorption assimilation  are sensitive treatment on particular individual.                 It has been said : It is wise to consider all non-surgical treatment options before resorting to an artificial hip or knee joint implant," said EULAR President Professor Iain B. McInnes, University of Glasgow, Scotland. Low calcium intake contributes to diminished bone density, early bone loss and an increased risk of fractures. Eating disorders. Severely restricting food intake and being underweight weakens bone in both men and women. There's no cure for osteoporosis, but proper treatment can help protect and strengthen your bones. These treatments can help slow the breakdown of bone in your body, and some treatments can spur the growth of new bone. In addition to causing permanent pain, osteoporosis causes some patients to lose height. When osteoporosis affects vertebrae, or the bones of the spine Weaken.Osteoarthritis is the most commonly diagnosed type of joint arthritis disease, which can affect hands, knees and hips. Knee arthritis is known to affect joint functionality causing knee pain and even leading to disability as it progresses. There are different stages of knee osteoarthritis (OA).The Center for Disease Control and Prevention has found that the number of people suffering from knee pain disorder is gradually rising.OA pain is easily identifiable through diagnostics and common symptoms. Some people who suffer from immense osteoarthritis knee pain may only show mild changes on x-ray
Lifestyle considerations like regular exercise may also prove to be helpful. Typically, the area around the knee joints will feel stiff and uncomfortable, particularly when sitting for an extended period, after rising in the morning, or after a workout. Though the cartilage and soft tissues remains at a healthy size, there is proteolytic breakdown of the cartilage matrix from an increased production of enzymes, such as metalloproteinase.
When your physician detects and diagnoses OA at this early stage, it is easier to follow a plan to stop the progression of this joint disease. , where there is obvious erosion to the cartilage surface between bones and fibrillation narrows the gap between the bones. There are proteoglycan and collagen fragments released into the synovial fluid as the disease progresses, wherein the bones develop spurs at the joints as it becomes rougher.With the progression of osteoarthritis of the knee, there is obvious joint inflammation which causes frequent pain when walking, running, squatting, extending or kneeling. Along with joint stiffness after sitting for long or when waking up in the morning, there may be popping or snapping sounds.
weight loss program, typically lasts six months ; which may help a little. Severe OA is considered  when  the joint space between the bones are considerably reduced, causing the cartilage to wear off, leaving the joint stiff. The breakdown of cartilage leads to a chronic inflammatory response, with decreased synovial fluid that causes friction, greater pain and discomfort when walking or moving the joint.
There is increased production of synovial metalloproteinases, cytokines and TNF that can diffuse back into the cartilage to destroy soft tissue around the knee. The advanced stage of the disease shows development of more spurs causing excruciating pain, which makes even everyday chores, including walking and descending stairs a challenge.
In cases of severe OA of the knee, an  orthopedic surgeon cuts the bone above or below the knee to shorten the length and help realign it for less stress on the knee joint. This surgery helps protect the knee by shifting the weight of the body away from the site of the bone spur growth and bone damage.

Another surgical option is total knee replacement, or arthroplasty. During this surgical procedure, the damaged joint is removed and replaced with a plastic or metal prosthesis device. Recovery from surgery may take several weeks and requires patience and discipline, with continuous physical and occupational therapy to regain full mobility.

Clinical Nutrition & Dietetics:. There are specific recommendations in regard to nutritional supplements and botanical medicines. Osteoarthrosis (OA) is sometimes referred to as osteoarthritis, degenerative arthritis, or degenerative joint disease. It is characterized by degeneration of the articular cartilage and subchondral bone. It can result in joint deformities, with characteristic Bouchard’s nodes in the proximal phalangeal joints of the fingers and Heberden’s nodes in the distal joints of the fingers. The predictable symptoms include joint pain and stiffness, becoming more chronic over time. Other symptoms commonly included joint swelling, weakness and/or paresthesias in the extremities and decreased range of motion. Typically, joints in the fingers, thumb, knees, spine, and hips can be involved. It can also be the result of trauma to a single joint leading to degeneration of the joint years later. Abnormal cartilage, congenital deformities of joint(s), infection of a joint, crystal deposition and/or other arthritic conditions can lead to OA of the joint. The knee is the most common joint to be affected.
Conventional treatment includes non-steroidal anti-inflammatory drugs. While their use is widespread, there is some evidence that long-term use may inhibit the synthesis of the cartilage matrix and accelerate the deterioration of cartilage. However, there is some evidence that is contradictory. There are some studies that demonstrate that the anti-inflammatory effects of low-dose aspirin slow the progression on cartilage loss in OA.  Additionally, there are significant risks associated with the use of NSAIDs; including gastrointestinal bleeding and cardiovascular events.
Musculoskeletal joint derangement and misalignments have been demonstrated to play a role in the development of OA. Joint alignment, muscle strength nutritional status, previous trauma and mid-stance loads all seem to contribute to the development of OA, Swimming and isometrics have been identified as superior to other types of exercise for the treatment of OA.  Conversely, strengthening the muscles controlling the hip and knee is an effective way to mitigate some of the disabling symptoms associated with OA in the lower extremity.
Diet should focus on the decrease of inflammation, as well as improving insulin sensitivity and achieving ideal body weight. Obesity is a major risk factor for the development of OA in the knees and hips. There is considerable evidence linking metabolic syndrome with inflammatory cytokines implicated in OA. Insulin stimulates chondrocytes and chondroblasts to secrete proteoglycans, which are essential to the proper function of cartilage. Decreased sensitivity to insulin has a detrimental effect on cartilage. Combining weight loss and exercise is known to decrease the risk of developing OA, as well as reducing the symptoms of OA and improving function. General guidelines for diet include a plant-based diet, relying on fresh fruits and vegetables, as well as whole grains, fatty fish and lean meats. Dairy products and gluten-containing grains are known to be allergenic in many people, and therefore should be minimized or eliminated from the diet. Certain individuals may benefit from eliminating nightshades (tomatoes, white potatoes, peppers, and eggplant) from the diet, most likely because of an individual’s sensitivity to these plants. One study looked at Diosgenin, a steroidal saponin found in nightshades. Diosgenin possesses diverse biological activities including anti-inflammatory properties. It has been shown to inhibit the synthesis of inflammatory mediators, such as prostaglandin E2 and interleukins. Therefore, there is a possibility that these plants may actually be beneficial for OA.
Antioxidants:The intake of carotenoids, ascorbate and vitamin E have all been demonstrated to improve the symptoms of OA and even to reverse the progression of the disease. The odds of having hip OA were reduced by nearly two fold when the individual consumed recommended or higher amounts of vitamin C. It has been demonstrated that vitamin C protects chondrocytes from oxidative damage from hydrogen peroxide by regulating multiple regulatory pathways. Astaxanthin, a carotenoid found in a variety of marine mammals and plants, has been shown to reduce Matrix Metalloproteinases (MMP), a substance known to degrade cartilage. The carotenoids in saffron, namely crocins and crocetin, are known to prevent the expression of MMP due to IL.Vitamin D:According to several studies, low plasma levels of vitamin D are associated with an increased risk of OA. However, there are some studies that contradict these findings. Improved outcomes following knee arthroplasty were noted after vitamin D supplementation.  Vitamin D has been shown to have a positive effect on the mass of the quadriceps muscles. It seems that inadequate levels of vitamin D contribute to the progression of OA, but it is not clear whether supplementation is beneficial in improving symptoms in those afflicted with the disease.
Vitamin K:Low vitamin K status is associated with an increased incidence of OA. The anti-inflammatory effects of vitamin K are thought to play an important role in the prevention of OA. The carboxylation of osteocalcin is dependent on vitamin K. Vitamin K deficiency caused serum undercarboxyled osteocalcin levels to increase.                                                                                  So in principle of ancient ayurveda way of treatment is broadly considered;in treating well joint disorders.  I have found cartilaginous erosion injury have been relieved. Synovial effusion stopped. Knee Joint strengthened. Ayurveda has explained rasayana for nutrition in different diseases and  at conditions of necessity ; which has got importance in treatment.       

                                                  

FOR CONSULTATIONS CONTACT:                         91-6362583334, 91- 9972831762,                           Dr. Ramachandra B H                                      BAMS(Bachelor in Ayurveda medicine & surgery),BAMS(Emergency Allopathic medicine),PGDYS (Post Graduate Diploma in Yogic Science )Former District Ayush officer .